Homology to NEPHRIN Lacking NEPH1, a Novel Protein with Proteinuria and Perinatal Lethality in Mice

نویسندگان

  • Brian P. Zambrowicz
  • David R. Powell
  • Michael Starbuck
  • Mary Brandt
  • Anupma Gupta
  • Alexander Turner
  • Robert Geske
  • Charles A. Montgomery
  • James P. Barrish
  • Brian N. Mathur
  • Dorit B. Donoviel
  • Deon D. Freed
  • Hannes Vogel
  • DORIT B. DONOVIEL
  • DEON D. FREED
  • HANNES VOGEL
  • DAVID G. POTTER
  • EDITH HAWKINS
  • JAMES P. BARRISH
  • BRIAN N. MATHUR
  • ROBERT GESKE
  • CHARLES A. MONTGOMERY
  • MICHAEL STARBUCK
  • MARY BRANDT
  • ANUPMA GUPTA
  • RAMIRO RAMIREZ-SOLIS
  • BRIAN P. ZAMBROWICZ
  • DAVID R. POWELL
چکیده

10.1128/MCB.21.14.4829-4836.2001. 2001, 21(14):4829. DOI: Mol. Cell. Biol. Ramirez-Solis, Brian P. Zambrowicz and David R. Powell Michael Starbuck, Mary Brandt, Anupma Gupta, Ramiro Alexander Turner, Robert Geske, Charles A. Montgomery, C. Potter, Edith Hawkins, James P. Barrish, Brian N. Mathur, Dorit B. Donoviel, Deon D. Freed, Hannes Vogel, David G. Homology to NEPHRIN Lacking NEPH1, a Novel Protein with Proteinuria and Perinatal Lethality in Mice

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Neph1 and nephrin interaction in the slit diaphragm is an important determinant of glomerular permeability.

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Neph1 Is Reduced in Primary Focal Segmental Glomerulosclerosis, Minimal Change Nephrotic Syndrome, and Corresponding Experimental Animal Models of Adriamycin-Induced Nephropathy and Puromycin Aminonucleoside Nephrosis

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تاریخ انتشار 2001